Methods: Rats with acetic acid-induced gastric ulcer treated with JWD or cimetidine for 13 days, the apoptotic index ( AI)、 proliferating cell nuclear antigen labeling index ( LI) and cell cycle were detected. 方法:制备乙酸致大鼠胃溃疡模型,分别予健胃汤及西咪替丁治疗13d后,检测胃溃疡粘膜细胞凋亡指数(AI)、增殖细胞核抗原标记指数(LI),并分析细胞周期变化情况。
The inhibitive effects of gastric cancer cell-dendritic cell fusion vaccines on tumor cell proliferation cycle in vivo and in vitro were detected by flow cytometry. 流式细胞术检测融合疫苗对体外培养胃癌细胞周期及体内种植瘤组织细胞周期的影响。
Conclusion TGF α can increase the expression of cyclin E and D1 in gastric carcinoma cells to promote their cell cycle progress. 结论TGFα可显著促进胃癌细胞CyclinE和D1的表达,从而发挥其促细胞周期进程的作用。
Effect of C2 gene on humen gastric cancer cell cycle C2基因对人胃癌细胞周期的影响
Results: The mixture could reduce the damage index of gastric mucosa, increase the DNA content of gastric mucosa cells and raise the ratio of G1/ G0 cell cycle significantly. 结果:胶原肉桂油混合剂能降低胃粘膜损伤指数,增加胃粘膜细胞的DNA含量,提高G1/G0细胞周期的比例,实验组和对照组之间的差异有显著意义。
Analysis of gene expression profile of G1/ S transition in gastric cancer cell cycle 胃癌细胞周期G1/S转换期基因表达谱的分析
Analysis of gene expression profiles in gastric cancer cell cycle 胃癌细胞周期基因表达谱的变化
The Inhibitory Effects of EGCG on Proliferation of Human Gastric Cancer MGC-803 Cells Via Induction of Cell Cycle G_1 Phase Arrest and Its Molecular Mechanism EGCG诱导人胃癌MGC-803细胞G1期阻滞及其分子机制
Methods Effect of SP on SGC 7901 gastric cancer cell cycle and apoptosis were determined by FCM and the source of Ca 2+ during intracellular [ Ca 2+] I change, observed by laser scanning confocal microscopy. 采用激光扫描共聚焦技术观察海嘧啶对人胃癌细胞内[Ca2+]i的影响及[Ca2+]i变化时Ca2+的来源。
In order to build up a gastric carcinoma cell expressing human cyclin G2 gene stably, a useful tool for the research on the function of cyclin G2 in gastric carcinoma cell cycle regulation was made. 应用脂质体基因转染技术建立稳定表达CyclinG2基因的胃癌克隆细胞,为深入研究CyclinG2在胃癌细胞周期调控的作用和机制提供理想的生物学模型。
Its mechanism curing precancerous lesions of gastric cancer may lie in promoting apoptosis of precancerous cells, and control the cell cycle. 其治疗胃癌前病变的作用机制可能与促进胃癌前细胞的凋亡,调控其细胞周期有关。
SGC-7901 gastric cancer cell lines are established in vitro. By observing the Melittin on cell proliferation, cell cycle, calcium ion and effect of telomerase in SGC-7901, Discuss the mechanism of Melittin, which provide experimental foundation for development of anti-tumor drug. 本课题建立胃癌SGC-7901细胞株的体外模型,通过观察Melittin对SGC-7901细胞的增殖、周期、钙离子及端粒酶的影响,初步探讨Melittin的作用机制,为其开发为抗肿瘤的新药提供试验依据。
The roles of TWIST in the development and progression of gastric cancer may be associated with cell cycle, apoptosis, invasion, and angiogenesis mechanisms. TWIST可通过细胞周期、凋亡、侵袭及血管生成等机制在胃癌的发生发展中发挥作用。